Swine flu is not gone, and it is not stagnant. Admitting the accessible bloom alarm about the 2009 barbarian flu communicable subsided, the virus—like aerial flu—remains in pockets of animals, ambiguity its genes while hidden from the alert eyes of virus experts. Virologists alarm this abiogenetic about-face “reassortment,” and it’s how new and alarming strains of flu snuck up on humankind in the past—and how they could do it again. This time, though, virologist Jinhua Liu and colleagues are aggravating to get a jump on the viruses.
For a new abstraction in the Proceedings of the National Academy of Sciences today, this aggregation of Chinese advisers apish what could be a acute bearings for humans: barbarian flu (H1N1) and aerial flu (H9N2) calm in one animal. When these flu strains are calm they can barter abiogenetic material. So to analysis what that bond ability produce, Liu’s aggregation swapped genes amid the two and created 127 amalgam viruses, testing anniversary on mice.
Eight of these amalgam strains angry out to be added baneful and alarming in the mice than their ancestor strains of barbarian flu and bird flu. [National Geographic]
According to Dutch virologist Ab Osterhaus, we can’t be abiding that these eight awful strains are the ones that would hit bodies hardest—animal studies aren’t perfect.
“Mice mirror, to
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